DISEASES OF THE BLOOD SYSTEM

The section on diseases of the blood

231 

Etiology. Known for the role of septic infection and intoxication (benzene, arsenobenzol, sulfonamides, piramidon, bismuth, thiuragyl). The pathogenesis. The basis of the failure lamblasting apparatus bone marrow (granulopoiesis), hyporegenerative - delay ripening of maternal

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268 

Etiology. Plays the role of the fungus Fusorium sporotrichioedes that develops wintered on the field grasses. The pathogenesis. The poison generated in overwintered grain millet, buckwheat, wheat, rye and other cereals, is stability; the use of such grain as food in the form

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374 

Etiology. Wound, injury of the vessel during surgery, stomach ulcers, ectopic pregnancy, diseases, which are characterized by hemorrhagic diathesis, hemophilia and others, can cause profuse bleeding, which occurs a sharp anemia. The pathogenesis. Anemia that develops as a res

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231 

In some cases the cause of the disease cannot be established. Has a value of infestation a lentetsom, the so-called botticella anemia - Botkin's disease, and syphilis, pregnancy. In the pathogenesis of this suffering, Botkin plays the role of a violation of the Central regulati

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206 

The etiology is unknown. Seems to play a role all the same infection (viral); sometimes in the study of the flora of the mucous membrane of the throat (sore throat) detected spirally Plaut-Vincent. I The pathogenesis is unclear. The toxin selectively annoying reticulo-endothe

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268 

Aplastic anemia is a symptom that accompanies various pathological conditions. In aplastic anemia, the bone marrow is called "tracks", due to atrophic mucous his rebirth, which indicates a complete loss of hematopoietic function. The osteosclerosis, cancer, metastatic to the bo

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235 

Etiology. Total or Subtotal gastrectomy leads to the development hyporegenerative or gipoplasticheskaya anemia; in a few cases, marked the emergence of permitiendonos anemia.

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436 

Etiology and pathogenesis is not always uslovima. In some cases, the fever has been observed in patients suffering from tropical malaria. Sometimes fever occurs after treatment with Hina. Distributed mainly in low, swampy areas, where malaria foci.  

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337 

Etiology. Suffering develops on the soil malaria, syphilis, autointoxication. The pathogenesis. Plays the role of a toxic factor that is produced by the spleen (initially this struck spleen, secondary - liver, resulting is splenorenal atrophic cirrhosis of the liver). In the

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277 

The etiology is unknown. Sick exclusively women aged mostly between 30 and 40 years. The pathogenesis. It is based on a violation of hemoglobinopathy. Bone marrow, according to the biopsy, functionally complete - rich content normoblasts and erythroblasts, it rarely discover

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285 

The etiology is not known. On Botkina the basis of chlorosis - psychoneurosis. Chlorosis ill only girls, mainly in the period of occurrence. they reach sexual maturity. The pathogenesis of impaired hemoglobinopathy.  

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327 

The etiology is not known. The pathogenesis. It is based on hyperplastic processes in the field erythroblastosis the bone marrow. Biopsy of the bone marrow affects the wealth of young nucleated erythrocytes - erythroblasts; there is also a lot of figures of mitosis elements o

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194 

Etiology. rare family disease. Ill almost exclusively men; women, remaining for the most part healthy, transmit the disease to their offspring through the male line. The pathogenesis. The main znachenie has a slow time of blood coagulation associated with the lack of producti

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273 

Etiology. There's a risk of haemolytic substances contributing to failure of the bone marrow, producing defective erythrocytes (microcity - microporosity), characterized by a sharp decrease in the osmotic resistance due to the predominance among them young forms. There have bee

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369 

Acute leukemia in its histogenesis not differ from chronic. As well as chronic, acute leukemia occur as milosav and lymphadenosis.

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316 

The etiology is unknown. The pathogenesis. Lymphosarcoma is a true hemoblastosis. The starting point of the disease - lymphoblasts (hemocytoblasts) - reticular cells. Of the local process in the field of lymphatic glands gradually observed aggressive growth into adjacent tiss

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290 

The etiology is unknown: Among provoking the disease factors it should be noted infection (sepsis)and rarely injured. The pathogenesis. Leukemia is a systemic or generalized disease characterized by hyperplastic tumor process in the field lamblasting apparatus. When pumping M

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411 

The etiology is unknown. Assume that this disease is contagious and is often combined with tuberculosis. However, there is reason to relate it to the retikulez (Vlados, Krajewski). The pathogenesis. Chronic lymph system. In glands, along with necrotic foci (soft texture), it

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260 

Etiology and pathogenesis see Lymphedemas chronic.

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295 

The etiology is unknown. The pathogenesis. Multiple myeloma true tumor (hemoblastosis), the starting point are reticular cells in the bone marrow various parts of the skeleton.  

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629 

Etiology. A number of diseases accompanied by repetitive small blood loss: gastric ulcer and duodenal ulcer, stomach cancer, hemorrhoids, tumors of the female genital organs (fibroma, myoma of the uterus, and others), hemorrhagic diathesis. The pathogenesis. Depending on the

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272 

This group includes the following diseases: purpura simplex, s purpura. peliosis rheumatica, purpura abdominalis, purpura fulminans. Etiology and pathogenesis. Vascular toxicity anaphylactic origin, which are multiple hemorrhages in the skin and mucous membranes. Purpura whil

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244 

The etiology is unknown. The most likely cause of the disease is syphilis infection. Men get sick more often than women. The pathogenesis. The basis of hemolysis is the emergence of the cold hemolysins in the blood (serum) of patients due to the presence in it of peculiar aut

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282 

The etiology is unknown. Particular importance should be given to infection and trauma as precipitating bleeding factors. The pathogenesis. Crucial defeat of the vascular wall endothelium of capillaries and trombopenia contributing to the onset of bleeding.

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