POSTPARTUM (POST-ABORTION) INFECTIOUS DISEASES

In this section you will find all kinds of infectious diseases that can appear after a birth or an abortion. However, also here You will be able to find out their causes, signs, symptoms, and treatment of postpartum and post-abortion infectious diseases.

485 

The etiology. Infection streptostafilokokkovoe. The pathogenesis. The infection gets into the epididymis or by hematogenous or through pipes from the uterus (usually) reg continuitatem or lymphogenous way.  

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937 

Wrong expressing milk, insufficient hygienic maintenance of the mammary glands, poor maintenance, improper technique of feeding. The result is the formation of linear discontinuities and erosion of the epidermis, becoming deeper cracked nipples.  

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402 

A sharp bend of the uterus anteriorly or posteriorly in the presence of a slow return of its development in the postnatal period leads to delayed discharge.  

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451 

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412 


1011 

The etiology. Getting into the mammary gland mainly through the cracks of nipples (see) infection, most often staph. The pathogenesis. The infection spreads or moves dairy (interstitial mastitis) or lymphatic (parenchymatous mastitis), often at the same time both ways. In som

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574 

The etiology. The small delay of the placenta, in some cases as late manifestation of infection (strepto-staphylococcal) of varying intensity. The pathogenesis. The delay of part of the placenta, the uterus is badly reduced. In bacterial inflammation of the blood clots easily

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469 

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1404 

The etiology. Infection, mainly streptococcal. The pathogenesis. The bacteria cause inflammation on the inner surface of the postpartum uterus, which captures more or less extensive part of her or confined to individual sites. It occurs as an independent disease, and as one

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539 

The etiology. Infection, mainly streptococcal. The pathogenesis. Breeding bacteria on the inner surface of the uterus, primarily at the placental site, germinate available here placental clots of blood vessels, causing inflammation.

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602 

The etiology. The infection often streptococcal. The pathogenesis. The infection spread through the lymphatic ducts (usually) breaks from the vagina, cervix or the uterus itself, resulting in a more or less dense infiltration. Sometimes parametritis is formed as a metastasi

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467 

Parvicellular - inflammation of the entire pelvic tissue. The etiology of infection, most often streptococcal. Pathogenesis - see Parameters. The inflammatory process involves the entire pelvic tissue. In some cases, is an inflammatory reaction to the defeat of adjacent org

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952 

Pelviureteric, limited pelvic peritonitis. The etiology. Infection with Streptococcus in a mixture with various other types of pathogenic microbial flora, often with E. coli. The pathogenesis. Inflammation of the pelvic peritoneum, arising either due to infection through t

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437 

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864 

The etiology. Infection, predominantly streptococcal and staphylococcal. The pathogenesis. From the primary lesion in the genital organs (mainly) or outside their infectious beginning periodically enters the bloodstream and spreads it throughout the body. Formed stasis. Bact

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459 

The etiology. Infection (strepto - and staphylococci in the first place). Pathogenesis - see Pyosepticemia. If septicaemia is not formed of metastatic lesions. The body is flooded with a large number of microbes, their toxins, poisons it, provided the corresponding reaction

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575 

The divergence of the symphysis pubis as a result of birth trauma, edema and hematoma in this area with subsequent inflammation, mostly not of bacterial origin. In rare cases, inflammation develops as a result of penetration of pathogenic microbes, even less - as metastasis sep

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542 

One of the symptoms of postpartum uterine diseases. As an independent disease can be observed in the hyperextension uterine muscles on the grounds of arthrogryposis, multiple pregnancy, prolonged labor; tumors of the uterus (fibroids).  

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792 

Peritonitis in the postpartum period see as either normal (see Surgical diseases) or metastatic puerperal peritonitis. The etiology and pathogenesis - see Pyosepticemia.

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508 


533 


525 

The etiology. The damage to blood vessels strepto-staphylococci. The pathogenesis. As a result of changes (under the influence of infection) blood vessels (veins), blood flow and the blood itself is formed of inflammatory infiltration of the vessel walls and surrounding tiss

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469 

The etiology. The damage to blood vessels strepto-staphylococci. The pathogenesis. As a result of changes (under the influence of infection) blood vessels (veins), blood flow and the blood itself is formed of inflammatory infiltration of the vessel walls and surrounding tiss

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406 

The etiology. The damage to blood vessels strepto-staphylococci. The pathogenesis. As a result of changes (under the influence of infection) blood vessels (veins), blood flow and the blood itself is formed of inflammatory infiltration of the vessel walls and surrounding tis

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534 

Bacterial growth in the crotch seams, abrasion of the vaginal mucosa, sometimes the cervix with superficial necrosis and inflammation in deprived epithelial cover areas of the vaginal mucosa.

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