313 

Etiology. The absence or low content in the diet of vitamin B2 (Riboflavin). The pathogenesis. Dietary Riboflavin is connected to the body with phosphoric acid (fosfauriliruetsa) and acts as ariboflavinosis acid and Flannagan dinucleotide. Connecting further with protein, it

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260 

Etiology. The absence or low content in food protivopellagricescoe vitamin PP (nicotinic acid or its amide), as well as the absence of certain amino acids (tryptophan). The pathogenesis. The greatest number of cases occurs in spring and summer. The most significant practical

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314 

Etiology. The absence or low content in food protivotarannogo vitamin C (ascorbic acid). The pathogenesis. Predisposing moments are monotonous diet and nutrition with inadequate overall calories. Slightly different forms of the disease are zynga in newborns and older children

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271 

Etiology. The probable cause of the disease is the absence or the low content in the diet of folic acid (peraluminous acid). The pathogenesis. Moments that predispose to disease in a warm climate, are transferred amoebic and bacillary dysentery, chronic appendicitis, hemorr

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364 

Etiology. The absence or reduced content in the diet of vitamin a effects on the body. The pathogenesis. The disease is characterized by the following phenomena: 1) violations tempo adaptation, blindness, xerophthalmia and keratomalacia different stages of the same disease

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1286 

Etiology. The absence or low content in food protevorevmaticescoe vitamin (thiamine, aneurine). The pathogenesis. In the absence of the vitamin in the diet or insufficient intake or absorption is disturbed mainly carbohydrate metabolism in the body. There are several forms

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