ALAKIJA ALIMENTARY TOXIC

ALAKIJA ALIMENTARY TOXIC- QR

DISEASES OF THE BLOOD SYSTEM : - etiology, pathogenesis, symptoms and course of, recognition, prediction and prevention

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What is the ALAKIJA ALIMENTARY TOXIC and how it is treated?

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Etiology and pathogenesis ALAKIJA ALIMENTARY TOXIC

Etiology. Plays the role of the fungus Fusorium sporotrichioedes that develops wintered on the field grasses.

The pathogenesis. The poison generated in overwintered grain millet, buckwheat, wheat, rye and other cereals, is stability; the use of such grain as food in the form of bread, cereal, pancakes, etc. can cause poisoning. It should be noted hypersensitivity to poison blood-forming organs, gastrointestinal tract and nervous system. As a result of intoxication indicates a progressive braking operation lamblasting apparatus that receives expression in the presence of leukopenia (neutropenia) - Alakai; however, with the passage of time involved in a pathological process platelet apparatus and erythropoiesis. Painting defeat the bone marrow matches aplasia. Hemorrhagic diathesis should be associated with impaired vascular membrane of the capillaries, resulting in increased their permeability, and the presence of thrombopenia. On the background radiation in patients with marked development of angina necrotic process. Trophic disorders (necrosis, gangrene of the mucous membranes, skin, and so on) are in connection with violations on the part of the nervous system.

Symptoms and course ALAKIJA ALIMENTARY TOXIC

Symptoms. Initial symptoms occur after taking substandard food (grain poisoning); salivation, burning sensation in the mouth, tongue and throat, dysphagia, pain on swallowing. In severe cases of poisoning, depending on the reactivity of the patient's body, there are, in addition to thrush, symptoms of gastroenteritis (nausea, vomiting, diarrhea, and others). One of the cardinal symptoms of this suffering is leukopenia (neutropenia). In the absence of appropriate therapeutic measures against a background of progressive anemia and leukopenia (decrease in white blood cell count below 1 LLC 1 mm3) noted the development of the final symptoms of the disease: necrotizing tonsillitis, fever (febris intermittens or continua), exacerbation of hemorrhagic diathesis, progressive anemia and sepsis.

Course and complications. The disease is divided into three stages:

1) acute gastro-intestinal tract (stomatitis, gastritis and so on);

2) leukopenia and hemorrhagic rash;

3) necrotic angina and acute exacerbation of hemorrhagic phenomena with increasing anemia.

Recognition ALAKIJA ALIMENTARY TOXIC

In some cases, developing sepsis. Objectively, lethargy, pallor, the presence of petechial in the form of purpura rash on the trunk and extremities. When pressed with your finger on the elements of the rash last not disappear. The most valuable diagnostic symptom is leukopenia (neutropenia). The presence of necrotic angina associated with leukopenia (neutropenia) and relevant anamnestic data (consumption of poor quality grain) quickly helps to make a diagnosis. The diagnosis helps and the fact endemic spread suffering.

Predictions ALAKIJA ALIMENTARY TOXIC

Prediction favorable with early intervention, i.e., the respective supply and treatment.

Prevention ALAKIJA ALIMENTARY TOXIC

Most important, along with early detection of endemi has early intervention and explanatory work among the population of the affected area to stop eating overwintered grain.

 

Treatment.

In the early stages of poisoning enough stopping power substandard food, laxatives and destination full of diety, rich in vitamins (fresh herbs). The time factor in the sense of the term intervention from the beginning poisoning plays in the success of the treatment is crucial. In an advanced stage of the disease, in addition to nutritious food (eggs, milk, curd, cheese, meat, fresh herbs, vitamins C, a, b-complex), transfusion of blood at a dose of 100 ml 2 - 3 times a week.

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